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Patients with a malignant esophageal obstruction caused by cancer present a challenging clinical problem. Conventional methods of palliative chemotherapy, such as Cisplatin, have failed to improve the overall survival in patients with severe, malignant esophageal obstruction, despite providing symptomatic relief in selected patients. This study reviewed all patients with a malignant esophageal obstruction resulting in death from malignant disease, in order to ascertain the response of their cancer to palliative radiotherapy. Thirty-six patients were treated with palliative radiotherapy between January 2000 and December 2003. The majority of the patients were referred from other centres for palliative radiotherapy and referred symptoms included dyspnoea (86%), chest pain (66%), dysphagia (55%), odynophagia (44%), haematemesis (38%), gastrooesophageal reflux (30%) and hiccups (13%). The median overall survival of all patients was 2 weeks. One patient survived an average of 3.2 weeks following radiotherapy. Esophageal carcinoma was the most common tumour and all patients had previously received or were receiving chemotherapy. Palliative radiotherapy appears to have no definitive effect on the survival of patients with a malignant esophageal obstruction resulting in death from cancer.Cell proliferation and survival in the lung in relation to lung structure.
Two fundamental questions in lung biology are how the structural and functional relationships of the normal lung are maintained, and how these relationships may be perturbed leading to lung disease and cancer. Lung structural relationships include alveolar and vascular size and shape; the number and morphology of alveolar type II and bronchiolar Clara cells; the presence of alveolar and bronchiolar epithelial cells (alveolar and bronchiolar’stem cells’); the presence of airway epithelial sub-mucosal glands and some interstitial cells (fibroblasts); and the presence of blood and lymphatic vessel capillaries. In this review the studies which have been undertaken to determine what role cell proliferation and survival play in the maintenance of normal lung structural relationships are discussed. The available evidence suggests that the alveolar type II cell is more important than the Clara cell in determining alveolar size and shape, and that alveolar
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